The Neuron and Neural Firing

Elijah Flores
7 min read
Listen to this study note
Study Guide Overview
This study guide covers neural communication, focusing on neurons, glial cells, and the spinal cord reflex arc. It details the neural transmission process, including resting potential, action potential, and reuptake. Key neurotransmitters and hormones are discussed, along with their functions. The guide also explains the effects of psychoactive drugs (agonists, antagonists, reuptake inhibitors) on neurotransmitters and behavior, including tolerance and addiction. Finally, it provides practice questions and exam tips.
#AP Psychology: Neural Communication - Your Final Review š§
Hey! Let's make sure you're totally prepped for the exam. This guide is designed to be your go-to for a quick, confident review. Let's dive in!
#1. The Building Blocks: Neurons and Glial Cells
#Neural Cells in Behavior
- Neurons: The stars of the show! They transmit info using electrical and chemical signals. Think of them as tiny messengers. āļø
- Form complex networks to process and relay information.
- Glial Cells: The unsung heroes! They support, insulate, and protect neurons. Like the stage crew for the neurons. š
- Provide structure and scaffolding for neural networks.
- Insulate neurons to enhance signal transmission.
- Facilitate communication between neurons.
- Transport waste products away from neurons.
Together, neurons and glial cells are the foundation for all behavior and mental processes. Sensory input, processing, decision-making, and motor output all rely on them working together.
#Spinal Cord Reflex Arc
- Reflex Arc: A super-fast, automatic response to a stimulus. Think of it as a shortcut. ā”
- Example: Pulling your hand away from a hot stove.
- Sensory Neurons detect the stimulus (heat).
- Interneurons in the spinal cord relay the signal.
- Motor Neurons cause muscles to react.
- This happens without the brain's direct involvement for super-fast reactions.
#2. The Neural Transmission Process
#Neural Transmission Mechanisms
- Resting Potential: Neuron is chillin', with a negative charge inside. š§
- Threshold: A stimulus makes the neuron less negative, reaching a critical point. Like flipping a switch.
- All-or-Nothing Principle: Once the threshold is reached, the neuron fires completely or not at all. No half-measures here.
- Depolarization: The action potential! The inside of the cell briefly becomes positive. š„
- Refractory Period: A short rest period where the neuron can't fire again. Gotta recharge.
- Reuptake: Neurotransmitters are reabsorbed to end the signal and prep for the next one. Like cleaning up after a party.
Don't confuse depolarization and repolarization. Depolarization is when the cell becomes more positive, while repolarization is when the cell returns to its negative resting potential.
- Disruptions can lead to disorders:
- Multiple Sclerosis: Damages myelin, impairing signal transmission. Think of it like a frayed wire.
- Myasthenia Gravis: Antibodies attack acetylcholine receptors, weakening muscles. Like a broken key in a lock.
š« Exclusion Note: Sodium-potassium pump is not on the AP exam. Don't sweat it!
#Neurotransmitter Functions
- Neurotransmitters: Chemical messengers that relay signals between neurons. Each has a specific job. šÆ
- Excitatory: Increase the chance of firing.
- Glutamate: The main excitatory neurotransmitter in the brain.
- Norepinephrine: Increases arousal and alertness. Get ready to go!
- Inhibitory: Decrease the chance of firing.
- GABA: The main inhibitory neurotransmitter, balancing excitation.
- Serotonin: Regulates mood, sleep, and appetite. Feel good!
- Some can be both excitatory and inhibitory, depending on the receptor.
- Dopamine: Motivation, reward, and motor control. The "feel good" neurotransmitter.
- Acetylcholine: Muscle contractions, learning, and memory. Key for movement and recall.
- Other key neurotransmitters:
- Endorphins: Natural pain relievers and euphoria-inducers. Like a natural high.
- Substance P: Transmits pain signals. Ouch!
- Excitatory: Increase the chance of firing.
Think of GABA as "Gotta Be Calm" (inhibitory) and Glutamate as "Get Going" (excitatory).
š« Exclusion Note: Only these neurotransmitters are on the AP exam. Focus here!
#Hormones in Behavior
- Hormones: Chemical messengers produced by glands, traveling through the bloodstream. Like long-distance messengers. š
- Adrenaline (Epinephrine): "Fight-or-flight" response. Get ready to react!
- Leptin: Signals satiety (fullness). Time to stop eating!
- Ghrelin: Stimulates hunger. Time to eat!
- Melatonin: Regulates sleep-wake cycles. Time to sleep!
- Oxytocin: Promotes social bonding and trust. Feel the love!
š« Exclusion Note: Only these hormones are on the AP exam. No need to memorize others.
#3. Psychoactive Drugs and Behavior
#Drug Effects on Neurotransmitters
- Agonists: Mimic or enhance neurotransmitter effects. Like a key that fits the lock perfectly.
- Opioids (heroin, morphine) are agonists for endorphins.
- Antagonists: Block neurotransmitter effects. Like a key that doesn't fit the lock.
- Caffeine is an antagonist for adenosine.
- Reuptake Inhibitors: Block reabsorption, prolonging effects. Like keeping the neurotransmitter in the synapse longer.
- Antidepressants (Prozac) are serotonin reuptake inhibitors (SSRIs).
#Psychological and Physiological Effects
- Stimulants: Increase neural activity and arousal.
- Caffeine and Nicotine: Mild stimulants.
- Cocaine and Amphetamines: Potent stimulants, can cause euphoria and paranoia.
- Depressants: Decrease neural activity, have sedating effects.
- Alcohol: Impairs judgment, coordination, and memory.
- Benzodiazepines (Xanax, Valium): Treat anxiety and insomnia.
- Hallucinogens: Cause perceptual and cognitive distortions.
- Marijuana (THC): Alters perception, judgment, and memory.
- LSD and Psilocybin: Induce vivid hallucinations.
- Opioids: Powerful pain relievers, induce euphoria and relaxation.
- Prescription Opioids (OxyContin, Vicodin): Treat severe pain.
- Heroin: Illegal opioid, high risk for overdose and addiction.
Stimulants speed up the nervous system, while depressants slow it down. Easy to remember!
#Drug Tolerance and Addiction
- Tolerance: Need higher doses for the same effect. The brain adapts, requiring more to get the same feeling.
- Addiction: Compulsive use despite negative consequences. The brain's reward system gets hijacked.
- Withdrawal: Symptoms when stopping drug use. The brain readjusts to the absence of the drug. Can be tough!
Be sure to understand the difference between agonists and antagonists, and how they affect neurotransmitter activity. This is a common point in both MCQs and FRQs.
#Final Exam Focus
- High-Priority Topics: Neural transmission, neurotransmitters, drug effects, and the nervous system's basic structure.
- Common Question Types: MCQs on neurotransmitter functions, FRQs on drug effects and the neural transmission process.
- Time Management: Quickly identify the core concept in each question. Don't overthink the obvious.
- Common Pitfalls: Confusing agonists and antagonists, mixing up neurotransmitters, and not understanding the basic neural communication process.
- Strategies: Connect concepts to real-life examples. Use mnemonics and analogies to remember key terms.
#
Practice Question
Practice Questions
#Multiple Choice Questions
-
Which neurotransmitter is primarily associated with the regulation of mood, sleep, and appetite? (A) Dopamine (B) Serotonin (C) GABA (D) Acetylcholine
-
A drug that blocks the reuptake of a neurotransmitter would have what effect? (A) It would decrease the amount of neurotransmitter in the synapse. (B) It would increase the amount of neurotransmitter in the synapse. (C) It would prevent the neurotransmitter from binding to the postsynaptic receptor. (D) It would cause the neuron to become hyperpolarized.
-
The action of pulling your hand away from a hot stove is an example of: (A) A voluntary response controlled by the brain. (B) A learned behavior. (C) A reflex arc controlled by the spinal cord. (D) A response mediated by hormones.
#Free Response Question
Scenario: A college student is struggling with anxiety and has started using a new medication prescribed by their doctor. The medication is designed to increase the activity of GABA in their brain. They also report using caffeine to stay awake during late-night study sessions.
(a) Explain how the prescribed medication is designed to reduce the student's anxiety. (b) Describe the mechanism of action of caffeine and its effects on the nervous system. (c) Discuss the potential long-term effects of using both the prescribed medication and caffeine.
Scoring Breakdown:
(a) (2 points) - 1 point for correctly identifying GABA as an inhibitory neurotransmitter. - 1 point for explaining that increasing GABA activity will decrease neural activity, reducing anxiety.
(b) (2 points) - 1 point for stating that caffeine is an antagonist for adenosine. - 1 point for explaining that blocking adenosine leads to increased neural activity and alertness.
(c) (3 points) - 1 point for mentioning the potential for tolerance with the prescribed medication. - 1 point for mentioning the potential for dependence or addiction with the prescribed medication. - 1 point for mentioning the potential for increased anxiety, jitters, and sleep disturbances from caffeine.
Alright, you've got this! Remember to stay calm, read each question carefully, and trust your knowledge. You are totally prepared! Go crush that exam! šŖ
Explore more resources

How are we doing?
Give us your feedback and let us know how we can improve